Heart resources

Module · Heart

FUSIC Heart resources

50 scans, 10 directly observed, all competencies. The five FUSIC Heart questions: LV dilated/impaired? RV dilated/impaired? Low venous return? Pericardial effusion? Pleural effusion?

Standard views & probe techniquePLAX, PSAX, A4C, subcostal, IVC — getting consistent images every time.

Use the phased-array (sector) probe, 2–4 MHz, for all cardiac views. Confirm indicator orientation by tapping the probe edge — it should match the screen marker.

  • PLAX: 3rd–4th left intercostal space, just left of sternum. Indicator to right shoulder. Shows LV, LVOT, aortic root, LA, MV, pericardium. Depth 14–16 cm.
  • PSAX: Rotate 90° clockwise from PLAX. Indicator to left shoulder. Fan from base (AV “Mercedes Benz”) through mid-cavity (papillary muscles) to apex.
  • A4C: Apex beat position, 5th–6th ICS, MCL. Indicator to left hip. All four chambers visible. Fan to A5C to see LVOT.
  • Subcostal: Subxiphoid, probe flat, indicator to patient’s left. Best in ventilated patients and post-arrest. Best pericardial view.
  • IVC view: Rotate to longitudinal from subcostal, indicator to head. Measure 2 cm proximal to RA, just distal to hepatic vein confluence.
If one window is poor, try another. Ventilated patients often have better subcostal windows. Left lateral decubitus improves parasternal views.
LV size & systolic functionQualitative grading, fractional shortening, FAC — practical bedside assessment.

FUSIC Heart uses qualitative (eyeball) assessment. Biplane Simpson’s volumes are FUSIC HD territory.

Qualitative grading:

  • Normal: Brisk inward motion; endocardium thickens clearly; cavity nearly obliterates at end-systole.
  • Mildly impaired: Reduced but definite inward motion.
  • Moderately impaired: Clearly reduced motion; LV does not contract well.
  • Severely impaired: Minimal motion; large dilated LV barely changes size.

Fractional shortening (FS): M-mode cursor through LV in PLAX at chordal level. FS = (LVEDD − LVESD) / LVEDD × 100. Normal ≥25%. <20% = significantly impaired.

FAC (A4C): Trace endocardial border at ED and ES. FAC = (EDA − ESA) / EDA × 100. Normal ≥35%.

LV size Men (LVEDD) Women (LVEDD)
Normal <5.6 cm <5.2 cm
Mildly dilated 5.6–6.0 cm 5.2–5.7 cm
Grossly dilated >6.5 cm >6.1 cm
FUSIC threshold: LVIDd >6 cm = grossly dilated. Visual assessment is the primary method.
A hyperdynamic LV (small cavity, brisk motion) in a shocked patient is NOT reassuring — it suggests hypovolaemia, vasodilation, or LVOTO. Do not call it normal function.
RV size & functionTAPSE, RV:LV ratio, D-shaped septum — the acutely stressed right heart.

Assess RV in A4C view — not from PLAX alone. The RV is the most frequently missed abnormality in critical care echo.

RV size (A4C): RV:LV end-diastolic area ratio. >0.6 = dilated. Ratio approaching 1:1 = severely dilated.

D-sign (PSAX at papillary level): D-shaped LV = RV pressure overload. Flat septum throughout systole = pressure overload. Flattening only in diastole = volume overload.

TAPSE: M-mode through lateral tricuspid annulus in A4C. Measure annular excursion. Normal ≥17 mm. <17 mm = impaired RV function.

McConnell’s sign: preserved apical RV motion with severe free-wall hypokinesis. Highly specific for acute PE in the right clinical context.
Acutely dilated, hypokinetic RV + shocked patient = consider massive PE, severe ARDS, or RV failure. Fluid may worsen RV dilatation — be cautious.
IVC & venous returnWhat the IVC tells you — and its significant limitations.

Measure IVC ~2 cm from RA entry, just proximal to hepatic vein confluence, in subcostal longitudinal view.

IVC finding Estimated RAP
<2.1 cm + collapses >50% Low (0–5 mmHg)
>2.1 cm + collapses <50% Elevated (>10 mmHg)

Collapsibility index (spontaneous breathing): (IVC max − IVC min) / IVC max × 100. >50% = likely fluid responsive.

Distensibility index (ventilated): (IVC max − IVC min) / IVC min × 100. >12% suggests responsiveness — only valid with no spontaneous breaths and TV ≥8 ml/kg.

IVC alone is an unreliable predictor of fluid responsiveness. Interpret alongside LV/RV assessment and clinical context. A normal IVC does not mean fluid is safe to give.
Pericardial effusion & tamponadeRecognition, sizing, and the echo signs of tamponade physiology.

Echo-free space surrounding the heart, outside the pericardial line. Best seen in PLAX and subcostal views.

Size: Small <1 cm (posterior only); Moderate 1–2 cm; Large >2 cm; Very large >2 cm + swinging heart.

Tamponade signs:

  • RV free-wall collapse in early diastole (most sensitive)
  • RA collapse for >1/3 of systole
  • Dilated IVC with <50% respiratory collapse
  • Swinging heart in large effusion
Tamponade is a clinical diagnosis — echo confirms it. A large effusion without haemodynamic compromise is not tamponade. A small localised post-surgical haematoma can cause tamponade.

Pericardial vs pleural: In PLAX, pleural effusion lies posterior to the descending aorta; pericardial lies anterior to it. The descending aorta is the landmark.

Pleural effusion from cardiac windowsIdentifying costophrenic fluid from PLAX and subcostal views.

From PLAX: pleural fluid lies posterior to the descending thoracic aorta as an echo-free space. Collapsed or consolidated lung may be seen floating in large effusions (“jellyfish sign”).

The descending aorta is the single most useful landmark for distinguishing pericardial from pleural fluid — a bright, round cross-section in PLAX.

For thorough lung assessment including effusion sizing and guided drainage, see FUSIC Lung resources.
Severe valvular diseaseRecognition only — features that should trigger expert referral.

FUSIC Heart does not require quantification of valve disease. Recognise the pattern and refer.

  • Aortic stenosis: Thickened, calcified, immobile AV leaflets in PLAX. Turbulent colour jet through restricted orifice in A5C/PSAX.
  • Aortic regurgitation: Diastolic colour jet into LVOT in PLAX/A5C.
  • Mitral stenosis: Doming leaflets in PLAX (“hockey stick”), restricted opening, LA enlargement.
  • Mitral regurgitation: Systolic colour jet into LA in A4C/PLAX. Severe = large turbulent jet filling >40% of LA.
Any significant valvular abnormality should be documented and a formal Level 2 echo requested. Your role is to recognise and escalate.
Pitfalls & artefactsCommon image traps and how to avoid them.
  • Reverberation behind pericardium: Bright parallel lines — can mimic effusion. True effusion is echo-free and moves with the heart; reverb stays fixed.
  • Epicardial fat: Anterior to RV, inside pericardium. Grey/echogenic, not echo-free. Stays anterior and does not cross the descending aorta landmark.
  • Near-field clutter: Fuzzy bright signal in first ~1 cm. Reduce gain or increase depth. Can obscure RV free wall.
  • Foreshortened apex in A4C: LV looks spherical — move probe medially and caudally.
  • Over-reading hyperdynamic LV as normal: Small cavity with brisk motion in a shocked patient = likely vasodilated or hypovolaemic. Not a reassuring finding.
  • Pericardial vs pleural: Use descending aorta as landmark in PLAX.

External reading